Search results for "Perforant path"

showing 7 items of 7 documents

Synaptic Phospholipid Signaling Modulates Axon Outgrowth via Glutamate-dependent Ca2+-mediated Molecular Pathways.

2015

Abstract Altered synaptic bioactive lipid signaling has been recently shown to augment neuronal excitation in the hippocampus of adult animals by activation of presynaptic LPA2-receptors leading to increased presynaptic glutamate release. Here, we show that this results in higher postsynaptic Ca2+ levels and in premature onset of spontaneous neuronal activity in the developing entorhinal cortex. Interestingly, increased synchronized neuronal activity led to reduced axon growth velocity of entorhinal neurons which project via the perforant path to the hippocampus. This was due to Ca2+-dependent molecular signaling to the axon affecting stabilization of the actin cytoskeleton. The spontaneous…

0301 basic medicineCognitive NeuroscienceNeuronal OutgrowthHippocampusGlutamic AcidAxon hillockSynaptic Transmission03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicinePostsynaptic potentialmedicinePremovement neuronal activityAnimalsbioactive phospholipidsCalcium SignalingAxonearly synchronized activityCells CulturedPhospholipidsChemistryOriginal ArticlesEntorhinal cortexPerforant pathActin cytoskeletonAxonsCell biologyCa2+-signalingentorhinal–hippocampal formation030104 developmental biologymedicine.anatomical_structureaxon outgrowthnervous systemCalcium030217 neurology & neurosurgeryMetabolic Networks and PathwaysCerebral cortex (New York, N.Y. : 1991)
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Neurons of the dentate molecular layer in the rabbit hippocampus.

2012

The molecular layer of the dentate gyrus appears as the main entrance gate for information into the hippocampus, i.e., where the perforant path axons from the entorhinal cortex synapse onto the spines and dendrites of granule cells. A few dispersed neuronal somata appear intermingled in between and probably control the flow of information in this area. In rabbits, the number of neurons in the molecular layer increases in the first week of postnatal life and then stabilizes to appear permanent and heterogeneous over the individuals' life span, including old animals. By means of Golgi impregnations, NADPH histochemistry, immunocytochemical stainings and intracellular labelings (lucifer yellow…

Central Nervous SystemAnatomy and PhysiologyCell Countchemistry.chemical_compoundMolecular Cell BiologyComparative AnatomyNeuronsMultidisciplinaryNeuronal MorphologyPyramidal CellsQRAnimal ModelsAnatomyElectrophysiologymedicine.anatomical_structureNissl BodiesNissl bodysymbolsMedicineFemaleRabbitsCellular TypesResearch Articlemedicine.drugHistologyScienceNeurophysiologyBiologygamma-Aminobutyric acidsymbols.namesakeModel OrganismsDevelopmental NeuroscienceBiocytinmedicineAnimalsBiologyCell ShapeLucifer yellowStaining and LabelingDentate gyrusPerforant pathEntorhinal cortexElectrophysiological PhenomenaNeuroanatomyElectrophysiologychemistrynervous systemCellular NeuroscienceDentate GyrusBiophysicsNeural Circuit FormationNeurosciencePLoS ONE
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Long-Term Potentiation in the Recurrent Inhibitory Circuit of the Dentate Gyrus

1988

The question of whether long-term potentiation occurs in the inhibitory circuits of the hippocampus remains controversial. Buszaki and Eidelberg (1982), recording extracellularly from putative interneurones (basket cells) in the dentate gyrus and area CAl of the anaesthetized rat, found a prolonged increase in probability of cell firing to afferent stimulation after high-frequency stimulation of Schaffer-commissural fibres, and concluded that LTP occurs at excitatory feedforward synapses onto interneurones. Similarly, Kairis et al (1987) have presented field potential evidence for LTP in feedforward synapses onto inhibitory neurones in the dentate gyrus of the anaesthetized rat. In the hipp…

Chemistrymusculoskeletal neural and ocular physiologyDentate gyrusHippocampusLong-term potentiationPerforant pathInhibitory postsynaptic potentialGranule cellmedicine.anatomical_structurenervous systemBasket cellmedicineExcitatory postsynaptic potentialNeuroscience
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Effects of histamine on dentate granule cells in vitro

1990

Abstract Hippocampal slices from rat brain were exposed to histamine and related substances in a perfusion chamber. Granule cells of the dentate gyrus were studied with conventional extra- and intracellular recording and a single electrode voltage clamp. Histamine caused, through activation of H 2 -receptors, a small depolarization, an increase in the number of synaptic and action potentials, a block of the long lasting (but not the early) component of spike afterhyperpolarizations and a reduction of the accommodation of action potential firing. These effects were mimicked by forskolin (suggests activation of adenylate cyclase). In voltage clamp, histamine blocked a long lasting calcium-dep…

General NeuroscienceDentate gyrusColforsinHistaminergicAction PotentialsRats Inbred StrainsIn Vitro TechniquesPerforant pathInhibitory postsynaptic potentialHippocampusMembrane PotentialsRatschemistry.chemical_compoundmedicine.anatomical_structurechemistryHistamine H2 receptormedicineExcitatory postsynaptic potentialBiophysicsAnimalsHistamine H3 receptorNeuroscienceHistamineAdenylyl CyclasesHistamineNeuroscience
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Different electrophysiological actions of 24- and 72-hour aggregated amyloid-beta oligomers on hippocampal field population spike in both anesthetize…

2010

Diffusible oligomeric assemblies of the amyloid beta-protein (Abeta) could be the primary factor in the pathogenic pathway leading to Alzheimer's disease (AD). Converging lines of evidence support the notion that AD begins with subtle alterations in synaptic efficacy, prior to the occurrence of extensive neuronal degeneration. Recently, however, a shared or overlapping pathogenesis for AD and epileptic seizures occurred as aberrant neuronal hyperexcitability, as well as nonconvulsive seizure activity were found in several different APP transgenic mouse lines. This generated a renewed attention to the well-known comorbidity of AD and epilepsy and interest in how Abeta oligomers influence neu…

MaleAmyloidAmyloid betaHippocampusHippocampal formationMicroscopy Atomic ForceSettore BIO/09 - FisiologiaHippocampusRats Sprague-DawleyAtomic force microscopyAlzheimer's disease; Amyloid; Excitability; Oligomer; Atomic force microscopymental disordersAnimalsMolecular BiologyNeuronsAnalysis of VarianceExcitabilityAmyloid beta-PeptidesbiologyPerforant PathwayChemistryGeneral NeuroscienceDentate gyrusLong-term potentiationPopulation spikeAlzheimer's diseaseElectric StimulationPeptide FragmentsRatsElectrophysiologyElectrophysiologyOligomerbiology.proteinNeurology (clinical)NeuroscienceDevelopmental BiologyBrain research
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Increased hippocampal head diffusivity predicts impaired episodic memory performance in early Alzheimer's disease

2010

Recent neuroanatomical and functional neuroimaging studies indicate that the anterior part of the hippocampus, rather than the whole structure, may be specifically involved in episodic memory. In the present work, we examined whether anterior structural measurements are superior to other regional or global measurements in mapping functionally relevant degenerative alterations of the hippocampus in Alzheimer's disease (AD). Twenty patients with early AD (MMSE 25.7+/-1.7) and 18 healthy controls were studied using magnetic resonance and diffusion-tensor imaging. Using a regions-of-interest analysis, we obtained volumetric and diffusivity measures of the hippocampal head and body-tail-section …

MaleCognitive NeuroscienceHippocampusExperimental and Cognitive PsychologyHippocampal formationHippocampusBehavioral NeuroscienceAlzheimer DiseasePredictive Value of TestsFunctional neuroimagingmedicineHumansDementiaAge of OnsetEpisodic memoryAgedMemory Disordersmedicine.diagnostic_testPerforant PathwayMagnetic resonance imagingmedicine.diseaseMagnetic Resonance ImagingMental RecallFemaleAtrophyPsychologyNeuroscienceDiffusion MRINeuropsychologia
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Allocortical neurofibrillary changes in progressive supranuclear palsy.

1992

Silver techniques for intraneuronal cytoskeleton abnormalities (neurofibrillary tangles and neuropil threads) and extracellular A4-amyloid deposits were used to examine lesions of the cerebral cortex in six cases of progressive supranuclear palsy (three were mentally unimpaired and three showed moderate degrees of dementia). Deposits of A4-amyloid protein occurred in small numbers or were absent. Neurofibrillary tangles and neuropil threads were present in all cases and were largely confined to the allocortex. A characteristic pattern of changes was found in the entorhinal cortex. The three mentally unimpaired individuals had mild cortical changes virtually confined to the transentorhinal r…

MalePathologymedicine.medical_specialtyAmyloidSilver StainingHippocampusBiologyHippocampal formationHippocampusPathology and Forensic MedicineProgressive supranuclear palsyCellular and Molecular NeuroscienceAlzheimer DiseasemedicineHumansAgedCerebral CortexAllocortexBrainNeurofibrillary tangleNeurofibrillary TanglesMiddle AgedPerforant pathmedicine.diseaseEntorhinal cortexmedicine.anatomical_structureCerebral cortexFemaleNeurology (clinical)Supranuclear Palsy ProgressiveActa neuropathologica
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